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Progesterone and Solving Erectile Dysfunction (ED)

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I recently had a patient call me about the use of progesterone for Erectile Dysfunction or ED. He claimed that after 4 days of using topical progesterone use he began to have morning erections. I was completely taken by surprise. I had never heard of Progesterone being used for Erectile Dysfunction.

One of the main worries about having ED is that the small vessels in the penis are clogged in the same way the small arteries around the heart. When the small arteries are clogged in the heart, this starves the heart resulting in a heart attack. So ED could be a sign that you have artery disease that needs to be addressed.

Both the arteries around the heart, feeding the heart, coronary arteries, and the small arteries in the penis can easily be cleaned out using Esselstyn’s diet from “Preventing and Reversing Heart Disease”.  Esselstyn, MD has shown conclusively that going on a plant based diet can easily reverse heart disease in as little as 2 years. He had many patients that reversed their Heart Disease using diet alone.

But anyway back to the topic of Progesterone and solving Erectile Dysfunction. Here is a forum discussing using progesterone to treat erectile dysfunction.  Some researchers trained rats for 9 days with copulation experiences. 42.5% of the rats displayed excellent sexual performance. 17.5% of the rats had adequate performance. 40% of the rats with low or no sexual performance had lower progesterone levels compared to the high performing rats. The low/no sexual performance rats also had significantly reduced testosterone levels compared to the excellent/adequate rats. The authors concluded that progesterone may have been the limiting factor in promoting the sexual performance of rats.[1]

One patent filed by Steven Ferguson claimed that using a progesterone/testosterone cream, correcting the hormonal imbalance of progesterone and testosterone toward normal values, most men in the study were able to obtain normal erectile function.[2]

Progesterone is the mother hormone. Progesterone can be used to make more testosterone by the body. However, progesterone is way up at the beginning of the corticosteroid synthesis chart. This means you don’t have to be as careful dosing it as something that is at the very end of the synthesis chain like testosterone.

Reading the forums, some claim that progesterone made their gyneocmastia (man boobs) disappear. Others claim 

that progesterone made their gynecomastia worse. This is easy to understand.

Progesterone will interact with other hormone active chemicals or herbs that is put on the skin in minute amounts. Progesterone will balance out or oppose weak xenoestrogens. However, progesterone in the presence of a strong xenoestrogen will actually make the xenoestrogen worse. This is because the body to protect itself when taking in chronic xenoestrogen tries to shut down its sensitivity to estrogen. When you take progesterone, the estrogen receptors are woken up, and now, the xenoestrogens appear to be much much worse. This is why progesterone taken with strong xenoestrogens would make the gynecomastia worse.

Xenoestrogens are simply foreign estrogens. Xenoestrogens are chemicals and herbs that fit into the estrogen receptor. They mimic weirdly the effect of estrogen. Xenoestrogens do NOT appear on the hormone test.

Anyway, the solution is simple.

Buy any product from us and get a list of safe products to use. Use the products for 2-3 months. The use of any other products is a no-no. Then, use progesterone. This way the progesterone will not interact with anything that you put on the skin. Anything on the skin is 10 times in potency of the oral dose because it bypasses the liver and goes directly into the body. Progesterone is pretty safe, because as a baby you are exposed to high amount of progesterone in the womb. During one day of third trimester pregnancy, a woman make 400 mg of progesterone.

The question is where is the Erectile Dysfunction coming from? We know that our diet will cause clogging of the arteries, and arteries are in the penis. Is there a secondary reason as well?  Could it be that many of these xenoestrogens are causing the Erectile Dysfunction? We know that cotton seed has a toxic compound and likely a hormonal active compound called gossypol. Since the 1950s, researchers have known that gossypol caused male rats to become infertile.[3] Chinese researcher looked into gossypol as a possible form of birth control for men. Chinese Researchers found that gossypol interfered with the formation of sperm.[4] Here is an MD, Ph.D. that discusses the effect of gossypol on human fertility. Too much gossypol means no sperm. Recently, I noticed that the yummy potato chips I was eating could have been fried in cotton seed oil. Cotton seed oil is in all the main meats and dairy products because cotton seed oil is used to feed them. For men with low sperm counts, Bruce Semon, M.D. Ph.D. recommends changing the diet. He also sees women conceive and carry to term recovering from years of miscarriage. Here are some comments on the  book “Feast Without Yeast”.

Well, could it be that some of these hormone disruptors are creating Erectile Dysfunction? Cut out all xenoestrogens on the skin, and let's see. If you have any success, I want to know for my own education. Xenoestrogens act like strange estrogen. Xenoestrogens are being applied by men on their skin. Anything applied to the skin is 10 times in potency compared to what you eat.  By using your herbal shampoo, are you turning yourself into a woman?  Well, they can’t be good for you.

Notes

1. Alvarenga TA, Andersen ML, Tufik S, "Influence of Progesterone on Sexual Performance in Male Rats,” J Sex Med 2010; Jul;7(7):2435-44.

2. https://www.google.com/patents/US20070167418

3. Ambrose, A. M., and D. J. Robbins. Studies on the chronic oral toxicity of cottonseed meal and cottonseed pigment glands. J. Nutrition. 43:357-70. 1951.

4. Medrano, F. J., Andreu, J.M. Binding of gossyol to purified tubulin and inhibition of its assembly into microtubules. Eur. J. Biochem. 1986; 158(1):63-9.